P Palthera
Signalling Peptides

Kisspeptin-10

KP-10 / Metastin(45–54)

Kisspeptin-10 is a C-terminal of the KISS1 gene product that activates the KISS1R () . Published research spans cell-based signalling , knockout-mouse models, and human reproductive-endocrinology pharmacology studies, alongside emerging bone-biology research.

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Signalling Peptides
Classification
Endogenous KISS1-receptor agonist fragment
Research stage
Receptor signalling and reproductive endocrinology literature
Sequence
Tyr-Asn-Trp-Asn-Ser-Phe-Gly-Leu-Arg-Phe-NH2
Molecular weight
1302.5 Da

Snapshot

Key takeaways

A three-bullet snapshot before reading the full dossier.

  1. 01

    Ten- KISS1R .

  2. 02

    Studied across cell, knockout-mouse, and human reproductive-endocrinology pharmacology contexts.

  3. 03

    Emerging research extends into bone-cell biology (osteoclasts, osteoblasts).

Dossier overview

4

research areas

3

references

3

handling notes

01

Mechanism of action

Acts as an at KISS1R (). Research describes downstream activation of neurons in reproductive-axis models, and in bone-cell studies, GPR54-dependent signalling affecting BMP2 expression and Src dephosphorylation.

02

Research applications

  • KISS1R / signalling studies
  • Reproductive endocrinology research
  • Bone-cell (osteoclast / osteoblast) biology
  • Peptide comparison

03

Study references

Each profile cites a minimum of two peer-reviewed sources, with model type and reported sample size where the source provides it. Findings are model-specific and must not be extrapolated to therapeutic use.

Kisspeptin-10 binding to Gpr54 in osteoclasts prevents bone loss by activating Dusp18-mediated dephosphorylation of Src

2024

Li Z et al. · Nature Communications

Model
In vitro — primary osteoclasts (differentiation in triplicate); in vivo — Kiss1, Gpr54, and Dusp18 conditional/full knockout mice
Sample
Gpr54 cKO n=7/group (4F/3M); Kiss1 cKO n=6/group (3F/3M); Dusp18 KO n=5–7/group; OVX experiments n=6/group; in vitro biological triplicates

KP-10 binding to was associated with suppressed osteoclast activity and reduced bone loss via Dusp18-mediated Src dephosphorylation in the models used.

PMID 38346942 DOI 10.1038/s41467-024-44852-9

Kisspeptin-10 (KP-10) stimulates osteoblast differentiation through GPR54-mediated regulation of BMP2 expression and activation

2018

Son HE et al. · Scientific Reports

Model
In vitro — C3H10T1/2 mouse mesenchymal cells; GPR54⁻/⁻ cells
Sample
Not reported in abstract

KP-10 treatment was associated with increased osteogenic including BMP2 via -dependent signalling.

PMID 29391507 DOI 10.1038/s41598-018-20571-2

Metabolic regulation of kisspeptin — the link between energy balance and reproduction

2020

Navarro VM et al. · Nature Reviews Endocrinology

Model
Narrative review
Sample
N/A (review)

Reviewed how metabolic factors regulate kisspeptin neurons and the reproductive axis, integrating rodent and human research.

PMID 32427949 DOI 10.1038/s41574-020-0363-7

Evidence caveats

  • Reproductive-endocrinology human studies typically use short controlled pharmacology designs; chronic-administration human data are limited.
  • Bone-biology findings are early-stage and have not been confirmed in human trials.

04

Storage and handling

Maintain in controlled research storage with lot and preparation records available.

  • Record - context and sample preparation details.
  • Control light, temperature, and moisture exposure during storage.
  • Keep peptide records linked to batch documentation.

05

Related reading

education

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Methodology

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